Substances that promote lucid dreaming. How nutrition affects the development of superpowers

Generally speaking, the described methods of developing the ability to have lucid dreams do not require taking any chemicals in order to achieve...

It is recommended to take about 50 mg of vitamin B6 half an hour or right before bed. This dosage should not be exceeded, as there is information that prolonged overdose of B6 leads to nerve damage and muscle spasms, and subsequent recovery takes a long time. The recommended dose of vitamin B is 1000-2000 mcg, also just before bedtime. This is said to significantly increase the vividness and vividness of dreams and increase the likelihood of lucid dreams.

However, again, do not forget about the danger of an overdose: this can lead to orange or red skin staining. Caffeine may be helpful for experimenting with sleep-wake cycles. Try, for example, drinking a large mug of coffee in the middle of the night! However, first check your individual reaction to caffeine, as studies have shown that in many people it suppresses REM sleep.

However, if you manage to fall asleep after drinking a large drink of coffee or taking caffeine pills, then it is likely that the caffeine will work as a stimulant and cause your brain to switch to active state while the body is sleeping. The dreams that arise in this case are particularly vivid, and all that remains for you to do is to remember at the right moment that you are dreaming. For example, I saw my first lucid dream after drinking 5 or 6 cups of instant coffee at night.

As for alcohol, its suppressive effect on REM sleep is beyond anyone's doubt. However, some people note the appearance of a very vivid dreams when they take a nap in the morning the day after taking a significant dose of alcohol. This is probably due to the reaction of the body, which restores the cycle of sleep phases after a period of suppression.

There are interesting data concerning nicotine. If you're a smoker and don't mind messing with your stomach, try sleeping for six hours, then get up, smoke two cigarettes, and go back to bed, but now focus on visualization all the time. own hands(or use another method of direct entry into a dream known to you). Try not to move or disturb yourself in any other way, otherwise the nicotine will make you fully awake and the experience will fail. This exercise is said to work much better with the "nicotine attack" than without it. Another drug that affects dreams is nutmeg.

Some consider it a hallucinogen, but the dose that must be taken to cause hallucinations will lead to poisoning of the body and severe vomiting. The use of a smaller amount contributes to the emergence of very vivid and vivid dreams, sometimes for one to two weeks.

Now for some food.

It is noticed that certain types foods affect the lucidity of dreams, or at least their vividness and memorability. One of these products is milk. It contains amino acids that have been found to have a stimulating effect on the production of melatonin, a neurotransmitter (chemical messenger between nerve cells) associated with sleep and dreams.

Other foods mentioned include popcorn, cheese, fish and ice cream. The reasons for their effect on dreams have not been established, but perhaps everything is again connected with amino acids. Cold ice cream, in addition, helps to reduce the stomach, which affects general level mental arousal. It was also said that good effect chamomile tea has on dreams. But many who tried to constantly drink it before bedtime noted only a general improvement in the quality of sleep and a feeling of freshness after waking up, nothing more. Try it yourself - it won't hurt.


The term "lucid dreaming" was coined by Frederick van Eyden. Lucidity usually begins in the middle of sleep, when the dreamer realizes that the experience does not take place in physical reality, but is a dream. Often this realization is initiated by the dreamer when he notices an impossible or unlikely event in the dream, such as flying or meeting a dead person. Sometimes awareness comes without any specific dream prompt; a person may suddenly realize that he is in a dream. A minority of lucid dreams (about 10% according to the studies of LaBerge and colleagues) are the result of the return of REM sleep (with dreams) immediately after waking up with continuous reflective consciousness. Basic definition lucid dream requires only the awareness that you are dreaming. However, the quality of awareness can vary greatly. With a high level of awareness, a person realizes that everything that happens in a dream happens in his mind, that there is no real danger and that he is in his bed and will soon wake up. At a low level of awareness, a person may be aware to a certain extent that he is dreaming, which may be enough to start flying or to change what he does in a dream, but not enough to understand that people in a dream are a dream manifestation that the sleeper cannot receive any physical harm, or that he is actually in bed.

Substances and dreams

In the circumstances described above, it is highly recommended in this area to develop safe way helping dreamers achieve higher states of awareness in their dreams more frequently. For this, neurotransmitter modulators such as acetylcholinesterase inhibitors can be used. Several substances have been proposed to increase the likelihood of lucid dreaming, from vitamins to prescription drugs. There are several good scientific research confirming these assertions. Lucid dreams are very dependent on the placebo effect; believing that something will stimulate lucid dreaming is very effective. Many prescription drugs, as well as marijuana and alcohol, alter the sleep cycle, usually by suppressing REM sleep. This leads to a phenomenon called "REM backlash" where a person experiences intense long periods of REM after the substance has worn off. This may manifest as nightmares, or perhaps as lucid dreams, as the brain is very active at this point. Drugs from the LSD family, including psilocybin and tryptamines, actually stimulate REM sleep (at doses small enough to allow sleep), resulting in longer REM periods. Due to the potentially dangerous and illegal nature of these substances, their use is discouraged.

Acetylcholine and Lucid Dreaming

It is known in the scientific literature that acetylcholine and its agonist, as well as acetylcholinesterase and its inhibitors/antagonists, can affect REM and sleep itself 14). J. A. Hobson, in his 1988 book The Dreaming Brain, says: "The cholinergic mechanisms of the brainstem cause sleep and dreaming." It has also been found that microinjection of the acetylcholine agonist carbachol in certain areas of the brain, such as the pons, induces long periods of REM sleep, and that many of the neurons critical for REM sleep respond to acetylcholine. Neither Hobson nor other scientists discussed the possibility of altering cholinergic levels with acetylcholinesterase inhibitors as a means of enhancing memory recovery and awareness. Historically, there are many cultures in which naturally occurring substances have allegedly been found to alter dream awareness in some way. One such substance, calea cazatecci or "Dream Weed", according to a 1986 article in the Journal of Ethnopharmacology by Lilian Mayagoitia et al., is "a plant used by the Indians of Juntal origin in Mexico to receive divine messages in their dreams." The neuroactive substances from this herb are sesquiterpene lactones and, to the best of our knowledge, are not associated with acetylcholinesterase inhibition or acetylcholinesterase inhibitors.

Herbs and plants

Both ancient methodologies and modern popular medicine attribute "oneirogenic" or "hypnotic" properties to numerous herbs and other natural substances, including herbs such as valerian, wormwood, mallein, kava kava, kitta of Crete, St. John's wort, salvia divinorum, Scutellaria indica, licorice root, verbena, jasmine, honeysuckle, cabbage, bee pollen, catnip, hops, skulikup, mimosa, lavender, damiana, santania somnifera, passionflower, chamomile, cardamom, gotu kola, ginkgo biloba, ibogaine, verbena, rose, cinnamon, calendula, nutmeg, peppermint, holly, yarrow and anise . There is little scientific evidence to support these putative effects.

DMAE

Other supplements and/or medications also claimed to be associated with mindfulness include B vitamins, melatonin, DMAE, and the psychedelic dextromethorphan. Some of these substances may be addictive, poisonous, and/or illegal. Lotsof, in U.S. Patent No. 4,499,096, issued Feb. 12, 1985, states " Quick method to interrupt the syndrome drug addiction" by using medicinal product Ibogaine. According to the patent, this drug induces "an intensification of dreams or a hallucinatory effect." This claimed "oneirogenic" effect is due to the fact that it induces dream behavior while leaving the ego perspective relatively intact. As stated in the patent, this is more of a hallucinatory effect than a lucid dream effect. However, because of these hallucinatory and other side effects, this drug is illegal in the US and thus not a desirable sleep aid. This drug is also not considered related to acetylcholinesterase inhibitors or "drugs that improve mental capacity". DMAE (2-dimethylaminoethanol) is Chemical substance, which has been proposed for the treatment of a number of conditions, affecting the brain and central nervous system. It is supposed to work by increasing the production of the neurotransmitter acetylcholine, although this has not been proven. It is believed that DMAE improves memory and mood, improves intellectual functioning. There are several good clinical research confirming this assertion. Such substances are known as "cholinergics" due to the fact that they increase the level of acetylcholine. They are traditionally used to treat conditions such as Alzheimer's dementia, tardive dyskinesia, and Huntington's chorea. Because DMAE is considered cholinergic, it has been proposed for the treatment of these disorders, although placebo-controlled studies have produced essentially negative results. There is an ongoing debate about whether DMAE increases acetylcholine levels. However, Sergio, W. stated in the August 1988 edition of Medical Hypotheses in the article "The use of DMAE (2-dimethylaminoethanol) to induce lucid dreams" subjectively allowed him and his wife to experience heightened sleep lucidity. It is not clear from the article to what extent its purported results stem from a placebo response or any specific or cholinergic effect.

Other devices

Also well known in the art is a US patent. No. 5,507,716, awarded to LaBerge and colleagues on April 16, 1996, for equipment and methods used to induce lucid dreams in sleeping people. This patent uses a device to help people achieve lucid dreams by detecting and monitoring eye and head movements of sleeping people, where eye movement activity in the absence of head movement is used to indicate the presence of REM sleep. By applying sensory stimuli to REM sleepers, you can let them know they are dreaming. Keith M. T. Hearne illustrated and described his respiratory measuring instrument in 1983 (Pat. No. 4.420.001). His device sensed changes in the temperature of a person's breathing in the respiratory passage, or in the flow of air into and out of the respiratory passage. Thermistors have been used in an electrical circuit to detect temperature changes in human breathing. When the rate of these temperature changes reached a high predetermined level, the signals created in the electrical circuit initiated an audible sound to help bring the sleeping person out of an unpleasant dream, waking them up or helping them enter a lucid dream state.

Aetylcholinesterase inhibitors

None of the above descriptions describe a method for enhancing lucid dreaming that involves administering a class of acetylcholinesterase inhibitors. Therapeutic agents for Alzheimer's disease such as donepezil (Aricept®), rivastigmine (Exelon®), galantamine (Reminyl®, Nivalin®), tacrine and huperzine result in a low incidence of side effects such as insomnia and gastrointestinal symptoms At the same time, these drugs significantly improve cognitive dream lucidity, clarity, self-reflection, recall, control, quirkiness, and visual vividness. The new patent relates to the field of lucid dreaming and to improving dream recall and dream lucidity through memory enhancement, including a class of substances that include acetylcholinesterase inhibitors (AChEls). Lucid dreaming means dreaming in which you know you are dreaming. AChE inhibitors (AchEl) inhibit the normal metabolic inactivation of acetylcholine (ACh) by inhibiting the enzyme acetylcholinesterase (AChE), resulting in accumulation of Ach. AchEl is most commonly used to improve memory, especially in Alzheimer's patients. Ach well known also that he plays important role in REM and is therefore suggested to improve dreams and awareness. When investigating the efficacy of donepezil (Aricept®) as an agent to improve lucid dreaming and cognitive clarity during REM sleep in normal patients, the following experiment was conducted with following results: A randomized, double-blind, placebo-controlled crossover study was performed with ten normal volunteers selected for their good memory and interest in lucid dreaming (7 males, 3 females aged 22-55). Patients were required to collect dream content data and demonstrate other self-report measures for three nights separated by a drug elimination period of at least one week. At bedtime, they took capsules containing 0 (placebo), 5, or 10 mg decepil in a balanced order of three doses. After each spontaneous awakening for three nights, Ss assessed sleep content on a range of measures, including bizarreness, complexity and intensity, affect, cognitive awareness, clarity, and control. They also assessed sleep quality, insomnia, and the degree of any adverse impact. Nine of 10 Ss (90%) reported one or more lucid dreams on the night of the experiment, and only one S reported lucid dreaming on the placebo night. Cognitive clarity, awareness, recall, control, quirkiness, and visual vividness were all significantly improved with donepezil compared with placebo. Effects were generally associated with the 10 mg dose of doepisil, giving significantly more high levels of these variables than the 5 mg dose, which in turn resulted in significant increases over placebo. The likelihood of lucid dreaming for these three conditions increased from 0.031 for placebo to 0.429 for dopil 5mg and 0.754 for pipetyl 10mg. The odds ratio for the 10 mg dose versus placebo was 24.3 (p<0,001). Донепизил также был связан со значительно повышенной частотой паралича сна и 40% -ным увеличением оценочного времени сна в ночное время (176 против 126 мин в течение ночи 9,0 ч, р <0,05). Частота нежелательных явлений, особенно легкой бессонницы и желудочно-кишечных симптомов, была низкой и, в основном, ассоциировалась с более высокой дозой (два пациента сообщили о тошноте и один – о рвоте). Таким образом, по сравнению с плацебо, дозы Donepizil 5 и 10 мг значительно улучшают самооценку когнитивной ясности, саморефлексии и осознанности во сне. Эти эффекты могут быть дополнительно усилены комбинацией ингибиторов ацетилхолинэстеразы с ацетилхолиновыми предшественниками, агонистами и / или вызывающими осознанность электронными устройствами. Помимо данных, полученных с помощью пипецила, мы имеем данные на Exelon® (ривастигмин, доза 6-12 мг) и Галантамин (Reminyl®, Nivalin® 8-16 мг). Оба препарата работают так же хорошо, как Aricept®), но имеют меньшее количество побочных эффектов. Также был протестирован гиперзин с многообещающими результатами, но до сих пор не разработали соответствие дозы с Aricept® (но 5 мг донепизила более эффективны, чем 150 мкг гиперзина).

Other substances

In addition, there are reports of successful use of nicotine patches. There is also evidence in the scientific literature showing the initiation/intensification of REM sleep with arecoline. It should be noted that REM alteration, mindfulness-enhancing effects, may be due to a number of neurophysiological effects induced by these "nootropics" in addition to acetylcholinesterase inhibition. These include: 1. The use of a cholinergic agonist (eg, nicotine). 2. Muscarinic receptor agonist use: drugs that mimic the effect of ACh on muscarinic receptors: Inc: muscarinic (M1), M2 and nicotinic agonists. (such as arecoline or recoline, a muscarinic receptor agonist) 3. Use of a presynaptic receptor antagonist to activate the remaining acetylcholine neurons. (That is, there are desensitizing oligonucleotide sequences in rats that block muscarinic M2 (but not M4) receptors, increasing extracellular levels of acetylcholine. This effect (desensitizing nucleotide sequences are complementary to a messenger RNA sequence. When desensitizing DNA or RNA is added to a cell, they bind 4. Use of allosteric modulators (such as allosteric potentiating ligands) of acetylcholine and nicotinic receptors (drugs that interact with the receptor through binding sites other than those used for acetylcholine and nicotinic agonists and antagonists) Other strategies for potentiating acetylcholine function to improve dream lucidity, including the use of other classes of compounds that work in a similar way, should be apparent to those skilled in the art. Awareness-enhancing effects may be due to a number of neurophysiological effects induced by these "nootropics" in addition to acetylcholinesterase inhibition. They include:

    Use of a cholinergic agonist (eg, nicotine).

    Muscarinic receptor agonist use: drugs that mimic the effect of ACh on muscarinic receptors: Inc: muscarinic (M1), M2 and nicotinic agonists. (such as arecoline or recoline, a muscarinic receptor agonist)

    Use of a presynaptic receptor antagonist to activate the rest of the acetylcholine neurons. (That is, there are desensitizing oligonucleotide sequences in rats that block muscarinic M2 (but not M4) receptors, increasing extracellular levels of acetylcholine. This effect (desensitizing nucleotide sequences are complementary to a messenger RNA sequence. When desensitizing DNA or RNA is added to a cell, they bind with a specific messenger RNA molecule and inactivate it.

Substances that promote lucid dreaming

Lucidity usually begins in the middle of sleep, when the dreamer realizes that the experience does not take place in physical reality, but is a dream. The term "lucid dreaming" was coined by Frederick van Eyden. Lucidity usually begins in the middle of sleep, when the dreamer realizes that the experience does not take place in physical reality, but is a dream. Often this realization is initiated by the dreamer when he notices an impossible or unlikely event in the dream, such as flying or meeting a dead person. Sometimes awareness comes without any specific dream prompt; a person may suddenly realize that he is in a dream. A minority of lucid dreams (about 10% according to the studies of LaBerge and colleagues) are the result of the return of REM sleep (with dreams) immediately after waking up with continuous reflective consciousness. The basic definition of lucid dreaming only requires the awareness that you are dreaming. However, the quality of awareness can vary greatly. At a high level of awareness, the person is aware that everything that happens in a dream is happening in his mind, that there is no real danger, and that he is in his bed and will soon wake up. At a low level of awareness, a person may be aware to a certain extent that he is dreaming, which may be enough to start flying or to change what he does in a dream, but not enough to understand that people in a dream are a dream manifestation that the sleeper cannot receive any physical harm, or that he is actually in bed.

Substances and dreams

In the circumstances described above, it is highly recommended in the art to develop a safe way to help dreamers achieve higher states of lucidity in their dreams more frequently. For this, neurotransmitter modulators such as acetylcholinesterase inhibitors can be used. Several substances have been proposed to increase the likelihood of lucid dreaming, from vitamins to prescription drugs. There are some good scientific studies to support these claims. Lucid dreams are very dependent on the placebo effect; believing that something will stimulate lucid dreaming is very effective. Many prescription drugs, as well as alcohol, alter the sleep cycle, usually by suppressing REM sleep. This leads to a phenomenon called "REM backlash" where a person experiences intense long periods of REM after the substance has worn off. This may manifest as nightmares, or perhaps as lucid dreams, as the brain is very active at this point.

Acetylcholine and Lucid Dreaming

It is known in the scientific literature that acetylcholine and its agonist, as well as acetylcholinesterase and its inhibitors/antagonists, can affect REM and sleep itself 14). J. A. Hobson, in his 1988 book The Dreaming Brain, says: "The cholinergic mechanisms of the brainstem cause sleep and dreaming." It has also been found that microinjection of the acetylcholine agonist carbachol in certain areas of the brain, such as the pons, induces long periods of REM sleep, and that many of the neurons critical for REM sleep respond to acetylcholine. Neither Hobson nor other scientists discussed the possibility of altering cholinergic levels with acetylcholinesterase inhibitors as a means of enhancing memory recovery and awareness. Historically, there are many cultures in which naturally occurring substances have allegedly been found to alter dream awareness in some way. One such substance, calea cazatecci or "Dream Weed", according to a 1986 article in the Journal of Ethnopharmacology by Lilian Mayagoitia et al., is "a plant used by the Indians of Juntal origin in Mexico to receive divine messages in their dreams." The neuroactive substances from this herb are sesquiterpene lactones and, to the best of our knowledge, are not associated with acetylcholinesterase inhibition or acetylcholinesterase inhibitors.

Herbs and plants

Both ancient methodologies and modern popular medicine attribute "oneirogenic" or "hypnotic" properties to numerous herbs and other natural substances, including herbs such as valerian, wormwood, mallein, kava kava, kitta of Crete, St. John's wort, salvia divinorum, Scutellaria indica, Licorice Root, Verbena, Jasmine, Honeysuckle, Cabbage, Bee Pollen, Catnip, Hops, Skullcap, Mimosa, Lavender, Damiana, Santania Somnifera, Passiflora, Chamomile, Cardamom, Gotu Kola, Ginkgo Biloba, Ibogaine, Verbena, Rose, Cinnamon, marigold, nutmeg, peppermint, holly, yarrow and anise. There is little scientific evidence to support these putative effects.

Other supplements and/or medications also claimed to be associated with mindfulness include B vitamins, melatonin, DMAE, and the psychedelic dextromethorphan. Some of these substances may be addictive, poisonous, and/or illegal. Lotsof, US Pat. No. 4,499,096, issued Feb. 12, 1985, teaches a "Rapid Method for Interrupting an Addiction Syndrome" using the drug Ibogaine. According to the patent, this drug induces "an intensification of dreams or a hallucinatory effect." This claimed "oneirogenic" effect is due to the fact that it induces dream behavior while leaving the ego perspective relatively intact. As stated in the patent, this is more of a hallucinatory effect than a lucid dream effect. However, due to these hallucinatory and other side effects, this drug is illegal in the US and thus not a desirable sleep aid. This drug is also not considered to be related to acetylcholinesterase inhibitors or "mental enhancers". DMAE (2-dimethylaminoethanol) is a chemical that has been proposed for the treatment of a number of conditions affecting the brain and central nervous system. It is supposed to work by increasing the production of the neurotransmitter acetylcholine, although this has not been proven. It is believed that DMAE improves memory and mood, improves intellectual functioning. There are several good clinical studies to support this claim. Such substances are known as "cholinergics" due to the fact that they increase the level of acetylcholine. They are traditionally used to treat conditions such as Alzheimer's dementia, tardive dyskinesia, and Huntington's chorea. Because DMAE is considered cholinergic, it has been proposed for the treatment of these disorders, although placebo-controlled studies have produced essentially negative results. There is an ongoing debate about whether DMAE increases acetylcholine levels. However, Sergio, W. stated in the August 1988 edition of Medical Hypotheses in the article "The use of DMAE (2-dimethylaminoethanol) to induce lucid dreams" subjectively allowed him and his wife to experience heightened sleep lucidity. It is not clear from the article to what extent its purported results stem from a placebo response or any specific or cholinergic effect.

Other devices

Also well known in the art is a US patent. No. 5,507,716, awarded to LaBerge and colleagues on April 16, 1996, for equipment and methods used to induce lucid dreams in sleeping people. This patent uses a device to help people achieve lucid dreams by detecting and monitoring eye and head movements of sleeping people, where eye movement activity in the absence of head movement is used to indicate the presence of REM sleep. By applying sensory stimuli to REM sleepers, you can let them know they are dreaming. Keith M. T. Hearne illustrated and described his respiratory measuring instrument in 1983 (Pat. No. 4.420.001). His device sensed changes in the temperature of a person's breathing in the respiratory passage, or in the flow of air into and out of the respiratory passage. Thermistors have been used in an electrical circuit to detect temperature changes in human breathing. When the rate of these temperature changes reached a high predetermined level, the signals created in the electrical circuit initiated an audible sound to help bring the sleeping person out of an unpleasant dream, waking them up or helping them enter a lucid dream state.

Actylcholinesterase inhibitors

None of the above descriptions describe a method for enhancing lucid dreaming that involves administering a class of acetylcholinesterase inhibitors. Therapeutic agents for Alzheimer's disease such as donepezil (Aricept®), rivastigmine (Exelon®), galantamine (Reminyl®, Nivalin®), tacrine and huperzine result in a low incidence of side effects such as insomnia and gastrointestinal symptoms At the same time, these drugs significantly improve cognitive dream lucidity, clarity, self-reflection, recall, control, quirkiness, and visual vividness. The new patent relates to the field of lucid dreaming and to improving dream recall and dream lucidity through memory enhancement, including a class of substances that include acetylcholinesterase inhibitors (AChEls). Lucid dreaming means dreaming in which you know you are dreaming. AChE inhibitors (AchEl) inhibit the normal metabolic inactivation of acetylcholine (ACh) by inhibiting the enzyme acetylcholinesterase (AChE), resulting in accumulation of Ach. AchEl is most commonly used to improve memory, especially in Alzheimer's patients. Ach is also well known to play an important role in REM and is therefore suggested to improve dreams and awareness. In investigating the efficacy of donepezil (Aricept®) as a means to improve lucid dreaming and cognitive clarity during REM sleep in normal patients, the following experiment was performed with the following results: A randomized, double-blind, placebo-controlled crossover study was performed with ten normal volunteers, selected for their good memory and interest in lucid dreaming (7 men, 3 women aged 22-55). Patients were required to collect dream content data and demonstrate other self-report measures for three nights separated by a drug elimination period of at least one week. At bedtime, they took capsules containing 0 (placebo), 5, or 10 mg decepil in a balanced order of three doses. After each spontaneous awakening for three nights, Ss assessed sleep content on a range of measures, including bizarreness, complexity and intensity, affect, cognitive awareness, clarity, and control. They also assessed sleep quality, insomnia, and the extent of any adverse effects. Nine of 10 Ss (90%) reported one or more lucid dreams on the night of the experiment, and only one S reported lucid dreaming on the placebo night. Cognitive clarity, awareness, recall, control, quirkiness, and visual vividness were all significantly improved with donepezil compared with placebo. Effects were generally associated with the 10 mg dose of doepisil, giving significantly higher levels of these variables than the 5 mg dose, which in turn produced significant increases over placebo. The likelihood of lucid dreaming for these three conditions increased from 0.031 for placebo to 0.429 for dopil 5mg and 0.754 for pipetyl 10mg. The odds ratio for the 10 mg dose versus placebo was 24.3 (p

Other substances

In addition, there are reports of successful use of nicotine patches. There is also evidence in the scientific literature showing the initiation/intensification of REM sleep with arecoline. It should be noted that REM alteration, mindfulness-enhancing effects, may be due to a number of neurophysiological effects induced by these "nootropics" in addition to acetylcholinesterase inhibition. These include: 1. The use of a cholinergic agonist (eg, nicotine). 2. Muscarinic receptor agonist use: drugs that mimic the effect of ACh on muscarinic receptors: Inc: muscarinic (M1), M2 and nicotinic agonists. (such as arecoline or recoline, a muscarinic receptor agonist) 3. Use of a presynaptic receptor antagonist to activate the remaining acetylcholine neurons. (That is, there are desensitizing oligonucleotide sequences in rats that block muscarinic M2 (but not M4) receptors, increasing extracellular levels of acetylcholine. This effect (desensitizing nucleotide sequences are complementary to a messenger RNA sequence. When desensitizing DNA or RNA is added to a cell, they bind 4. Use of allosteric modulators (such as allosteric potentiating ligands) of acetylcholine and nicotinic receptors (drugs that interact with the receptor through binding sites other than those used for acetylcholine and nicotinic agonists and antagonists) Other strategies for potentiating acetylcholine function to improve dream lucidity, including the use of other classes of compounds that work in a similar way, should be apparent to those skilled in the art. Awareness-enhancing effects may be due to a number of neurophysiological effects induced by these "nootropics" in addition to acetylcholinesterase inhibition. They include:

Use of a cholinergic agonist (eg, nicotine).

Muscarinic receptor agonist use: drugs that mimic the effect of ACh on muscarinic receptors: Inc: muscarinic (M1), M2 and nicotinic agonists. (such as arecoline or recoline, a muscarinic receptor agonist)

Use of a presynaptic receptor antagonist to activate the rest of the acetylcholine neurons. (That is, there are desensitizing oligonucleotide sequences in rats that block muscarinic M2 (but not M4) receptors, increasing extracellular levels of acetylcholine. This effect (desensitizing nucleotide sequences are complementary to a messenger RNA sequence. When desensitizing DNA or RNA is added to a cell, they bind with a specific messenger RNA molecule and inactivate it.

Whether certain foods can affect your sleep has never been studied before. Professor of Psychiatry Thor Nielsen, Chancellor of Montreal's University of Dreams and Nightmares, set out to investigate the connection between products and dream content. In lucid dreams, the dream itself takes control of the actions and the immediate result. "Mindful Eating" is the name given to foods and supplements purported to improve dreams.

Sleep and dreams

According to the National Institute of Neuralgic Disorders, there are five developmental phases of sleep. During the first phase - a person begins to fall asleep, he has dreams, for example, visual images that do not form a single plot. Most of the sleep passes in the second phase, when eye movement stops and brain activity slows down. During the third and fourth phases, brain activity decreases to a delta frequency. Most dreams occur in the next phase, when your eyes are in constant motion, as if you were watching a movie. This happens 70-90 minutes after falling asleep. Most people spend about two hours dreaming.

Cheese and dairy products

Cheese and other dairy products are commonly believed to induce more vivid dreams. University professor Milena Corridge believes that there are chemicals in cheese that transmit signals to the brain. Comedian May Martin has experimented with cheese as a sleep catalyst and says brie cheese gives her wings in her sleep, and gorgonzola gives her immortality. Research conducted in 2005 confirms Martina's words - the sharper the cheese, the sharper the dreams. Studies conducted with 200 participants showed that blue cheese causes the strangest dreams.

Other products and supplements

Fruit juices, ice cream, popcorn, salty foods, and fish are the most common causes of more complex dreams. Biologically active additives, according to manufacturers, encourage memories in dreams. Reliable evidence about herbs, vitamins, and supplements suggests that their effectiveness remains in question. Most researchers are skeptical about these studies. However, researcher Kellogg III believes that supplementing with 100-250 mg of vitamin B6 just before bed makes dreams more vivid and exciting.

Tryptophan is an amino acid that converts to serotonin. Serotonin induces vivid dreams at higher levels. Tryptophan is found in chicken, salmon, lamb, eggs, white rice and milk. Cheddar cheese contains the most tryptophan.

According to many, apple cider vinegar causes the most vivid and realistic dreams. Also, such products include pizza, cabbage and various herbs (for example, valerian and chamomile extract).

What not to eat

Drinks such as coffee and various energy drinks are more likely to negatively affect the content of dreams, says psychiatrist James McFarlane, director of the MedSleep Clinic, Toronto. Research conducted by Australian scientists in September 1992 supports this theory in an article published in the International Journal of Psychology. A group of people who took Tabasco mustard before going to bed could not fall asleep for a long time, people experienced more negative emotions than usual. Possible reasons for this could be indigestion and increased body temperature associated with sleep disturbances. Garlic also causes nightmares and rather strange dreams, according to Livestrong.

Can foods influence the development of superpowers? It turns out yes! Attention! The result depends on what you eat!

1. How are nutrition and the development of superpowers related?
2. What kind of food has a beneficial effect on psychic abilities?
3. What products block the information channel?
4. How do psychics feel about eating meat?
5. Why is a personal approach to the development of superpowers needed?

How are nutrition and the development of superpowers related?

Became known!

The development of superpowers - clairvoyance, clairaudience, empathy, telepathy, telekinesis, pyrokinesis and other psychic abilities, depends on what the practitioner eats! This is what ancient Eastern teachings say.

How does food affect psychic¹ sensitivity and the ability to read information? The theory of vibrations explains everything! It also provides a scientific rationale for separate nutrition and product compatibility.

Some types of food can raise the level of body vibrations, while others, on the contrary, will block the free flow of energy and, thereby, block the intuitive channel.

What do yogis say?

“To acquire psychic abilities, a person must take care of himself as a whole, he must pay equal attention to his body, mind and spirit. These aspects are interconnected with each other, which means that taking care of the body and proper nutrition have a beneficial effect on mental activity and the level of spirituality.

The secrets of nutrition of yogis, which were unknown to the West for a long time, look for

The entire material world consists of energy, including food!

Food products can contain positive or negative energy, which, after eating them, is transferred to a person.

For example, if an animal experienced fear, then the energy of fear and horror will be present in its meat. If the plant has been treated with chemicals, they will enter the body and accumulate there.

Therefore, it is very important to monitor the quality of products, and if it is not possible to use only environmentally friendly products, then you need to increase the vibration of food.

What food has a beneficial effect on mental abilities?

To increase vibrations, it is better to choose natural fresh products that have been harvested recently, as they contain the maximum amount of pure positive energy.

These are, first of all, fresh vegetables and fruits. It is advisable to make sure that they are not processed with chemicals and do not contain GMOs. It can be watermelons, papayas, pineapples, apples, cherries, as well as all kinds of berries. From vegetables, you can choose all kinds of salads, cabbage, pumpkin, celery, carrots, etc.

Do not forget about grains, legumes and nuts, which have a high energy value and do not block energy channels. It is better to replace white rice with brown, and wheat bread with rye.

Antioxidants are an important part of the diet!

They increase immunity, cleanse blood vessels, rejuvenate the body and promote unhindered circulation of the energy flow. These are garlic, ginger, honey, cayenne pepper and other spices that are not only good for health, but also give food a delicious variety of flavors.

And of course it's green tea!

What products block the information channel?

Processed foods, semi-finished products, and those containing various preservatives and flavorings do not contribute to the enhancement of psychic abilities.

Yogis, psychics and mediums who are developing their superpowers recommend avoiding foods high in sugar, fat, salt and limiting the use of white flour baked goods.

Proven!

Smoking and drinking alcoholic beverages have an extremely negative effect on brain function. In the presence of such bad habits, the development of superpowers can be difficult. And it is contraindicated to carry out any practices in a state of alcoholic, toxic and other intoxication - it can be life-threatening!

How do psychics feel about eating meat?

Many psychics refuse to eat meat, especially red meat, for two reasons:

1. Meat takes a long time to digest, and it takes a lot of vital energy to digest it. Part of the energy that should be spent on brain activity, the body is forced to redirect to the breakdown of animal protein, and this deprives a person of mental strength.

2. It is believed that the residual energy of the animal is stored in the meat, and all the negative emotions experienced by the animal at the time of death remain in it. Such negative energy reduces energy vibrations and can block the ability to read information.

The development of superpowers is not only practice, it is a special way of life that includes absolutely all aspects.

Why is a personal approach to the development of superpowers needed?

Partial or complete rejection of animal products cleanses the body, consciousness, increases the level of spirituality and promotes the development of mental abilities. But!

But there is no one rule for everyone! It is important to consider individual characteristics. To do this, before deciding on a diet, you should consult a nutritionist. When compiling a diet, it is important to take into account all the contraindications that may be, and only then seriously engage in the development of superpowers.

Notes and feature articles for a deeper understanding of the material

¹ A psychic is a person who is believed to have the ability to extrasensory perception (

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